Drug efflux pump verapamil

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  • tuberculosis, resulting in intrabacterial drug accumulation
  • 6 in the absence of verapamil to 1
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  • The inhibition of these efflux pumps is expected to increase drug concentration into target cells, organelles and intracellular bacteria

    These included thioridazine and piperine, a derivative of black pepper used to enhance the bioavailability of coadminis-tered drugs [21]

    We evaluated efficacy of the efflux pump inhibitor (EPI) verapamil (VER) with fluconazole (FLC) against FLC-resistant (CaR) and -susceptible C

    Efflux pump gene expression was studied by RT-qPCR upon exposure to clarithromycin

    [Investigation of Efflux Pump Genes in Resistant Mycobacterium tuberculosis Complex Clinical Isolates Exposed to First Line Antituberculosis Drugs and Verapamil Combination] By testing verapamil analogs, we show that verapamil directly inhibits M

    However, the extent to which

    We show that verapamil, a known efflux pump inhibitor, which inhibits macrophage-induced rifampicin tolerance, also inhibits M

    Recently, it has been demonstrated that verapamil, an efflux inhibitor, can reduce bacterial drug tolerance caused by efflux pump activity when administered in combination with available antituberculosis agents

    Recent studies in macrophages and zebrafish show that inhibition of mycobacterial efflux pumps with verapamil reduces the bacterial drug tolerance and may enhance drug efficacy

    Other efflux pumps identified in P

    This multi-phase study investigated the contributions of efflux pumps to Mycobacterium tuberculosis drug resistance

    However, the final effect of inhibition of the pumps' activity was the same Discovering an NCE that inhibits antibiotic drugs' efflux via an efflux pump is a tedious process

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